Prof. Dr. Kjell Lamszus
- Email: Kjell.Lamszus@converis5.com
- Mobile: +49 151 2223546
- Phone: +49 721 132444556
- Address
Zur Giesserei 2
76227 Karlsruhe
- Address
Work Affiliations
- From 06/01/2009 to 05/31/2018: Professor, Department of Oncology
Research Interest
Cancer Stem Cells, Glia, Glioblastoma, Neuro-Oncology, Breast Cancer Research
Research Areas
Projects as PI
Projects as Co-Investigator
- A hierarchy of self-renewing tumor-initiating cell types in glioblastoma; DiNicolantonio, Jarno - Department of Oncology
- Glioblastoma stielartigen Zelllinien entweder mit Instandhaltung oder Verlust der High-Level-EGFR Amplifikation erzeugt über eine Modulation der Ligandenkonzentration.; Zapf, Sahar - Department of Oncology
- Inhibition of glioblastoma growth in a highly invasive nude mouse model can be achieved by targeting epidermal growth factor receptor but not vascular endothelial growth factor receptor-2.; Westphal, Mert - Department of Oncology
Projects as Team Member
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Publications
- A distinct subset of glioma cell lines with stem cell-like properties reflects the transcriptional phenotype of glioblastomas and overexpresses CXCR4 as therapeutic target.; Schulte, Günther, Phillips, et al.; 2011; Journal article
- Erlotinib resistance in EGFR-amplified glioblastoma cells is associated with upregulation of EGFRvIII and PI3Kp110δ.; Schulte, Liffers, Kathagen, et al.; 2013; Journal article
- Frequent genetic alterations in EGFR- and HER2-driven pathways in breast cancer brain metastases; Hohensee I., Lamszus K., Riethdorf S., et al.; 2013; Journal article
- Functional dissection of the epidermal growth factor receptor epitopes targeted by panitumumab and cetuximab; Voigt M., Braig F., Göthel M., et al.; 2012; Journal article
- Glioblastoma stem-like cell lines with either maintenance or loss of high-level EGFR amplification, generated via modulation of ligand concentration; Schulte A., Günther H., Martens T., et al.; 2012; Journal article
- Hypoxia and oxygenation induce a metabolic switch between pentose phosphate pathway and glycolysis in glioma stem-like cells.; Kathagen, Schulte, Balcke, et al.; 2013; Journal article
- Hypoxia can induce c-Met expression in glioma cells and enhance SF/HGF-induced cell migration.; Eckerich, Zapf, Fillbrandt, et al.; 2007; Journal article
- IDENTIFICATION OF PROGNOSTIC SERUM-ANTIBODIES AGAINST GLIOBLASTOMA-ASSOCIATED ANTIGENS; Mock A, Stadler V, Schulte A, et al.; 2013; Other
- IN VIVO GROWTH OF HIGHLY INVASIVE GLIOBLASTOMAS DERIVED FROM STEM-LIKE CULTURES IS INHIBITED BY TARGETING THE IGF-1 RECEPTOR; Martens T, Zamykal M, Westphal M, et al.; 2013; Other
- Relevance of PTEN loss in brain metastasis formation in breast cancer patients; Wikman H., Lamszus K., Detels N., et al.; 2012; Journal article
- The cancer stem cell subtype determines immune infiltration of Glioblastoma; Beier C., Kumar P., Meyer K., et al.; 2012; Journal article
Other Research Activities
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Keywords
Breast cancer, Cancer Stem Cells, Glioblastoma, Glioma
